clone d10d11 Search Results


clone d10d11  (Cell Signaling Technology Inc)
88
Cell Signaling Technology Inc clone d10d11
Clone D10d11, supplied by Cell Signaling Technology Inc, used in various techniques. Bioz Stars score: 88/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
https://www.bioz.com/result/clone d10d11/product/Cell Signaling Technology Inc
Average 88 stars, based on 1 article reviews
clone d10d11 - by Bioz Stars, 2026-02
88/100 stars
  Buy from Supplier
anti fip200 rabbit mab  (Cell Signaling Technology Inc)
96
Cell Signaling Technology Inc anti fip200 rabbit mab
a Volcano-plot obtained from microarray analysis of MDA-MB231 cells treated with CP-31398. The number of overexpressed and downregulated genes (F.C. > 2; p < 0.05) is indicated. b Genes belonging to the p53 pathway/network (hallmark_P53_pathway; GSEA) are enriched in overexpressed genes following CP-31398 treatment (24 and 48 h). c Targeted analysis of microarray results using a pre-established list of genes involved in autophagosomes formation. Most of these genes are upregulated following 24–48 h CP-31398 treatment. Genes indicated in red encode proteins belonging to the ULK complex. d RT-qPCR validation of ULK1, ULK2 , ATG101 , ATG13 , <t>FIP200</t> , ATG5, ATG12, GABARAPL2 , and ZFYVE1 expression following CP-31398 treatment (24 and 48 h) compared to untreated cells (F.C. fold-change). e CP-31398 treatment (24–48 h) increases the expression of several proteins belonging to the ULK complex (ULK1, ATG101, ATG13, FIP200). ATG5 was used as a control. The normalized ratio protein of interest/Actin was calculated by densitometry analysis and display. Data are representative of three independent experiments. f PFT-α (an inhibitor of p53 transcriptional activity) inhibits CP-31398-dependent induction of ULK1 expression, while ULK2 expression is not affected. Data ( d , f ) are the mean ± s.d. of five independent RT-qPCR experiments performed in duplicate. The p values ( d , f ) were determined by unpaired two-tailed Student's t test. ( NS non-significant)
Anti Fip200 Rabbit Mab, supplied by Cell Signaling Technology Inc, used in various techniques. Bioz Stars score: 96/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
https://www.bioz.com/result/anti fip200 rabbit mab/product/Cell Signaling Technology Inc
Average 96 stars, based on 1 article reviews
anti fip200 rabbit mab - by Bioz Stars, 2026-02
96/100 stars
  Buy from Supplier
rabbit mono anti-fip200  (Cell Signaling Technology Inc)
90
Cell Signaling Technology Inc rabbit mono anti-fip200

Rabbit Mono Anti Fip200, supplied by Cell Signaling Technology Inc, used in various techniques. Bioz Stars score: 90/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
https://www.bioz.com/result/rabbit mono anti-fip200/product/Cell Signaling Technology Inc
Average 90 stars, based on 1 article reviews
rabbit mono anti-fip200 - by Bioz Stars, 2026-02
90/100 stars
  Buy from Supplier
nfkb p50 monoclonal antibody 5d10d11  (Thermo Fisher)
N/A
NFkB p50 Monoclonal Antibody for Western Blot IF ICC IHC Flow
   Buy from Supplier
blk monoclonal antibody 9d10d1  (Thermo Fisher)
N/A
BLK Monoclonal Antibody for IHC
   Buy from Supplier
influenza a h5n1 a barnswallow hongkong d10 1161 2010 hemagglutinin ha gene orf cdna clone expression plasmid  (Sino Biological)
N/A
Full length Clone DNA of Influenza A H5N1 A barnswallow HongKong D10 1161 2010 Hemagglutinin
   Buy from Supplier

Image Search Results


a Volcano-plot obtained from microarray analysis of MDA-MB231 cells treated with CP-31398. The number of overexpressed and downregulated genes (F.C. > 2; p < 0.05) is indicated. b Genes belonging to the p53 pathway/network (hallmark_P53_pathway; GSEA) are enriched in overexpressed genes following CP-31398 treatment (24 and 48 h). c Targeted analysis of microarray results using a pre-established list of genes involved in autophagosomes formation. Most of these genes are upregulated following 24–48 h CP-31398 treatment. Genes indicated in red encode proteins belonging to the ULK complex. d RT-qPCR validation of ULK1, ULK2 , ATG101 , ATG13 , FIP200 , ATG5, ATG12, GABARAPL2 , and ZFYVE1 expression following CP-31398 treatment (24 and 48 h) compared to untreated cells (F.C. fold-change). e CP-31398 treatment (24–48 h) increases the expression of several proteins belonging to the ULK complex (ULK1, ATG101, ATG13, FIP200). ATG5 was used as a control. The normalized ratio protein of interest/Actin was calculated by densitometry analysis and display. Data are representative of three independent experiments. f PFT-α (an inhibitor of p53 transcriptional activity) inhibits CP-31398-dependent induction of ULK1 expression, while ULK2 expression is not affected. Data ( d , f ) are the mean ± s.d. of five independent RT-qPCR experiments performed in duplicate. The p values ( d , f ) were determined by unpaired two-tailed Student's t test. ( NS non-significant)

Journal: Cell Death & Disease

Article Title: The pharmalogical reactivation of p53 function improves breast tumor cell lysis by granzyme B and NK cells through induction of autophagy

doi: 10.1038/s41419-019-1950-1

Figure Lengend Snippet: a Volcano-plot obtained from microarray analysis of MDA-MB231 cells treated with CP-31398. The number of overexpressed and downregulated genes (F.C. > 2; p < 0.05) is indicated. b Genes belonging to the p53 pathway/network (hallmark_P53_pathway; GSEA) are enriched in overexpressed genes following CP-31398 treatment (24 and 48 h). c Targeted analysis of microarray results using a pre-established list of genes involved in autophagosomes formation. Most of these genes are upregulated following 24–48 h CP-31398 treatment. Genes indicated in red encode proteins belonging to the ULK complex. d RT-qPCR validation of ULK1, ULK2 , ATG101 , ATG13 , FIP200 , ATG5, ATG12, GABARAPL2 , and ZFYVE1 expression following CP-31398 treatment (24 and 48 h) compared to untreated cells (F.C. fold-change). e CP-31398 treatment (24–48 h) increases the expression of several proteins belonging to the ULK complex (ULK1, ATG101, ATG13, FIP200). ATG5 was used as a control. The normalized ratio protein of interest/Actin was calculated by densitometry analysis and display. Data are representative of three independent experiments. f PFT-α (an inhibitor of p53 transcriptional activity) inhibits CP-31398-dependent induction of ULK1 expression, while ULK2 expression is not affected. Data ( d , f ) are the mean ± s.d. of five independent RT-qPCR experiments performed in duplicate. The p values ( d , f ) were determined by unpaired two-tailed Student's t test. ( NS non-significant)

Article Snippet: From Cell Signaling: anti-caspase 3 (p35) mouse mAb (clone 3G2), anti-cleaved caspase 3 (p19/p17) rabbit mAb (clone 5A1E), anti-PARP rabbit pAb (cat #9542), anti-Atg101 rabbit mAb (clone E1Z4w), anti-Atg13 rabbit mAb (clone D4P1K), anti-FIP200 rabbit mAb (clone D10D11), anti-ULK1 rabbit mAb (clone D8H5), anti-AMPKα rabbit mAb (clone D5A2), anti-Phospho-AMPKα (Thr172) rabbit mAb (clone D4D6D), anti-mTOR rabbit pAb (cat #2972), anti-Phospho-mTOR (Ser2448) rabbit pAb (cat #2971), anti-LC3B rabbit pAb (cat #2775), anti-Atg5 rabbit mAb (clone D5F5U), anti-Bid rabbit mAb (cat #2002).

Techniques: Microarray, Quantitative RT-PCR, Biomarker Discovery, Expressing, Control, Activity Assay, Two Tailed Test

Journal: Developmental Cell

Article Title: CCPG1 Is a Non-canonical Autophagy Cargo Receptor Essential for ER-Phagy and Pancreatic ER Proteostasis

doi: 10.1016/j.devcel.2017.11.024

Figure Lengend Snippet:

Article Snippet: Rabbit mono anti-FIP200 (clone D10D11) - IB, IF, Peptide Array , Cell Signaling Technologies , 12436.

Techniques: Affinity Purification, Peptide Microarray, Control, Virus, Recombinant, Protease Inhibitor, Mass Spectrometry, Polymer, SYBR Green Assay, Stable Transfection, Expressing, Plasmid Preparation, Software, Microscopy